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教育部委託研究計畫      計畫執行:國立臺灣大學圖書館
 
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機構 日期 題名 作者
中國醫藥大學 2010-02 The c-Jun N-terminal Kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition. (Gao YJ); (Xu ZZ); (Liu YC); 溫永銳(Yeong-Ray Wen); (Decosterd I); (Ji RR)*
臺北醫學大學 2008 Activation of p38 mitogen-activated protein kinase in spinal microglia contributes to incision-induced mechanical allodynia 溫永銳; Wen YR; Suter MR; Ji RR; Wu YS; Kohno T; Wang KC; Sun WZ; Wang CC
中國醫藥大學 2007-08 Nerve conduction blockade in the sciatic nerve prevents but does not reverse the activation of p38 mitogen-activated protein kinase in spinal microglia in the rat spared nerve injury model. 溫永銳(Yeong-Ray Wen); (Suter MR); (Kawasaki Y); (Huang J); (Pertin M); (Kohno T); (Berde CB); (Decosterd I); (Ji RR)*
臺北醫學大學 2007 Do glial cells control pain? 溫永銳; Suter MR; Wen YR; Decosterd I; Ji RR
臺北醫學大學 2007 Nerve conduction blockade in the sciatic nerve prevents but does not reverse the activation of p38 mitogen-activated protein kinase in spinal microglia in the rat spared nerve injury model 溫永銳; Wen YR; Suter M; Kawasaki Y; Huang J; Pertin M; Kohno T; Berde C; Decosterd I; Ji RR
臺北醫學大學 2007 Role of the CX3CR1/p38 MAPK pathway in spinal microglia for the development of neuropathic pain following nerve injury-induced cleavage of fractalkine. 溫永銳; Zhuang ZY; Kawasaki Y; Tan PH; Wen YR; Huang J; Ji RR
中國醫藥大學 2007 Do glial cells control pain (Suter MR); 溫永銳(Yeong-Ray Wen); (Decosterd I); (Ji RR)*
臺北醫學大學 2006 A peptide JNK inhibitor blocks mechanical allodynia after spinal nerve ligation: respective roles of JNK activation in primary sensory neurons and spinal astrocytes for neuropathic pain development and maintenance 溫永銳; Zhuang ZY; Wen YR; Zhang DR; Borsello T; Bonny C; Strichartz GR; Decosterd I; Ji RR
臺北醫學大學 2006 Neural-glial interaction in the spinal cord for the development and maintenance of nerve injury-induced neuropathic pain. 溫永銳; Ji RR; Wen YR
中國醫藥大學 2006 Neural-glial interaction in the spinal cord for the development and maintenance of nerve injury-induced neuropathic pain. (Ji RR)*; 溫永銳(Yeong-Ray Wen)

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