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Taiwan Academic Institutional Repository >
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"lay jd"
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| 國家衛生研究院 |
2024-07-01 |
The role of AKR1B10 in lung cancer malignancy induced by sublethal doses of chemotherapeutic drugs
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Jang, TH;Lin, SC;Yang, YY;Lay, JD;Chang, CL;Yao, CJ;Huang, JS;Chuang, SE |
| 國家衛生研究院 |
2023-10-04 |
Intervention of AXL in EGFR signaling via phosphorylation and stabilization of MIG6 in non-small cell Lung cancer
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Yang, YY;Lin, SC;Lay, JD;Cho, CY;Jang, T;Ku, HY;Yao, CJ;Chuang, SE |
| 國家衛生研究院 |
2019-11 |
PTBP1-mediated regulation of AXL mRNA stability plays a role in lung tumorigenesis
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Cho, CY;Chung, SY;Lin, S;Huang, JS;Chen, YL;Jiang, SS;Cheng, LC;Kuo, TH;Lay, JD;Yang, YY;Lai, GM;Chuang, SE |
| 國家衛生研究院 |
2016-02 |
Negative feedback regulation of AXL by miR-34a modulates apoptosis in lung cancer cells
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Cho, CY;Huang, JS;Shiah, SG;Chung, SY;Lay, JD;Yang, YY;Lai, GM;Cheng, AL;Chen, LT;Chuang, SE |
| 國家衛生研究院 |
2014-02 |
Oxidative stress enhances AXL-mediated cell migration through an AKT1/Rac1-dependent mechanism [Erratum:Free Radical Biology and Medicine. 2013 Dec;65:1246-1256.]
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Huang, JS;Cho, CY;Hong, CC;Yan, MD;Hsieh, MC;Lay, JD;Lai, GM;Ann-Lii, C;Chuang, SE |
| 國家衛生研究院 |
2013-12 |
Oxidative stress enhances Axl-mediated cell migration through an Akt1/Rac1-dependent mechanism
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Huang, JS;Cho, CY;Hong, CC;Yan, MD;Hsieh, MC;Lay, JD;Lai, GM;Cheng, AL;Chuang, SE |
| 臺北醫學大學 |
2013 |
Oxidative stress enhances Axl-mediated cell migration through an Akt1/Rac1-dependent mechanism
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Huang, JS;Cho, CY;Hong, CC;Yan, MD;Hsieh, MC;Lay, JD;Lai, GM;Cheng, AL;Chuang, SE. |
| 國家衛生研究院 |
2008-09-18 |
Receptor tyrosine kinase AXL is induced by chemotherapy drugs and overexpression of AXL confers drug resistance in acute myeloid leukemia
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Hong, CC;Lay, JD;Huang, JS;Cheng, AL;Tang, JL;Lin, MT;Lai, GM;Chuang, SE |
| 臺北醫學大學 |
2008 |
Receptor tyrosine kinase AXL in induced by chemotherapy drugs and overexpresion of AXL confers drug resistance in acute myeloid leukemia.
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賴基銘; Hong CC; Lay JD; Huang JS; Cheng AL; Tang JL; Lin MT; Lai GM; Chuang SE |
| 國家衛生研究院 |
2007-09 |
Pathogenesis and mechanism of disease progression from hemophagocytic lymphohistiocytosis to Epstein-Barr virus-associated T-cell lymphoma: Nuclear factor-kappa B pathway as a potential therapeutic target
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Chuang, HC; Lay, JD; Hsieh, WC; Su, IJ |
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