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教育部委託研究計畫      計畫執行:國立臺灣大學圖書館
 
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機構 日期 題名 作者
國家衛生研究院 2024-07-01 The role of AKR1B10 in lung cancer malignancy induced by sublethal doses of chemotherapeutic drugs Jang, TH;Lin, SC;Yang, YY;Lay, JD;Chang, CL;Yao, CJ;Huang, JS;Chuang, SE
國家衛生研究院 2023-10-04 Intervention of AXL in EGFR signaling via phosphorylation and stabilization of MIG6 in non-small cell Lung cancer Yang, YY;Lin, SC;Lay, JD;Cho, CY;Jang, T;Ku, HY;Yao, CJ;Chuang, SE
國家衛生研究院 2019-11 PTBP1-mediated regulation of AXL mRNA stability plays a role in lung tumorigenesis Cho, CY;Chung, SY;Lin, S;Huang, JS;Chen, YL;Jiang, SS;Cheng, LC;Kuo, TH;Lay, JD;Yang, YY;Lai, GM;Chuang, SE
國家衛生研究院 2016-02 Negative feedback regulation of AXL by miR-34a modulates apoptosis in lung cancer cells Cho, CY;Huang, JS;Shiah, SG;Chung, SY;Lay, JD;Yang, YY;Lai, GM;Cheng, AL;Chen, LT;Chuang, SE
國家衛生研究院 2014-02 Oxidative stress enhances AXL-mediated cell migration through an AKT1/Rac1-dependent mechanism [Erratum:Free Radical Biology and Medicine. 2013 Dec;65:1246-1256.] Huang, JS;Cho, CY;Hong, CC;Yan, MD;Hsieh, MC;Lay, JD;Lai, GM;Ann-Lii, C;Chuang, SE
國家衛生研究院 2013-12 Oxidative stress enhances Axl-mediated cell migration through an Akt1/Rac1-dependent mechanism Huang, JS;Cho, CY;Hong, CC;Yan, MD;Hsieh, MC;Lay, JD;Lai, GM;Cheng, AL;Chuang, SE
臺北醫學大學 2013 Oxidative stress enhances Axl-mediated cell migration through an Akt1/Rac1-dependent mechanism Huang, JS;Cho, CY;Hong, CC;Yan, MD;Hsieh, MC;Lay, JD;Lai, GM;Cheng, AL;Chuang, SE.
國家衛生研究院 2008-09-18 Receptor tyrosine kinase AXL is induced by chemotherapy drugs and overexpression of AXL confers drug resistance in acute myeloid leukemia Hong, CC;Lay, JD;Huang, JS;Cheng, AL;Tang, JL;Lin, MT;Lai, GM;Chuang, SE
臺北醫學大學 2008 Receptor tyrosine kinase AXL in induced by chemotherapy drugs and overexpresion of AXL confers drug resistance in acute myeloid leukemia. 賴基銘; Hong CC; Lay JD; Huang JS; Cheng AL; Tang JL; Lin MT; Lai GM; Chuang SE
國家衛生研究院 2007-09 Pathogenesis and mechanism of disease progression from hemophagocytic lymphohistiocytosis to Epstein-Barr virus-associated T-cell lymphoma: Nuclear factor-kappa B pathway as a potential therapeutic target Chuang, HC; Lay, JD; Hsieh, WC; Su, IJ

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